Metabolism of selenite to selenosugar and trimethylselenonium in vivo: tissue dependency and requirement for S-adenosylmethionine-dependent methylation

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Standard

Metabolism of selenite to selenosugar and trimethylselenonium in vivo : tissue dependency and requirement for S-adenosylmethionine-dependent methylation. / Jackson, Matthew I; Lunøe, Kristoffer; Gabel-Jensen, Charlotte; Gammelgaard, Bente; Combs, Gerald F.

I: Journal of Nutritional Biochemistry, Bind 24, Nr. 12, 12.2013, s. 2023-30.

Publikation: Bidrag til tidsskrift › Tidsskriftartikel › Forskning › fagfællebedømt

Harvard

Jackson, MI, Lunøe, K, Gabel-Jensen, C, Gammelgaard, B & Combs, GF 2013, 'Metabolism of selenite to selenosugar and trimethylselenonium in vivo: tissue dependency and requirement for S-adenosylmethionine-dependent methylation', Journal of Nutritional Biochemistry, bind 24, nr. 12, s. 2023-30. https://doi.org/10.1016/j.jnutbio.2013.04.007

APA

Jackson, M. I., Lunøe, K., Gabel-Jensen, C., Gammelgaard, B., & Combs, G. F. (2013). Metabolism of selenite to selenosugar and trimethylselenonium in vivo: tissue dependency and requirement for S-adenosylmethionine-dependent methylation. Journal of Nutritional Biochemistry, 24(12), 2023-30. https://doi.org/10.1016/j.jnutbio.2013.04.007

Vancouver

Jackson MI, Lunøe K, Gabel-Jensen C, Gammelgaard B, Combs GF. Metabolism of selenite to selenosugar and trimethylselenonium in vivo: tissue dependency and requirement for S-adenosylmethionine-dependent methylation. Journal of Nutritional Biochemistry. 2013 dec.;24(12):2023-30. https://doi.org/10.1016/j.jnutbio.2013.04.007

Author

Jackson, Matthew I ; Lunøe, Kristoffer ; Gabel-Jensen, Charlotte ; Gammelgaard, Bente ; Combs, Gerald F. / Metabolism of selenite to selenosugar and trimethylselenonium in vivo : tissue dependency and requirement for S-adenosylmethionine-dependent methylation. I: Journal of Nutritional Biochemistry. 2013 ; Bind 24, Nr. 12. s. 2023-30.

Bibtex

@article{0d0c11cddbad488dbe40faa4c5b60777,
title = "Metabolism of selenite to selenosugar and trimethylselenonium in vivo: tissue dependency and requirement for S-adenosylmethionine-dependent methylation",
abstract = "Impaired S-adenosylmethionine (SAM)-dependent transmethylation and methylation capacity feature in diseases related to obesity or aging, and selenium (Se) metabolism is altered in these states. We tested the hypothesis that SAM metabolism is required for methylation and excretion of Se in a rat model. Four hours after selenite and periodate-oxidized adenosine (POA; an inhibitor of SAM metabolism) were administered, circulating markers of single-carbon status were unchanged, except for decreased circulating phosphatidylcholine (P",
author = "Jackson, {Matthew I} and Kristoffer Lun{\o}e and Charlotte Gabel-Jensen and Bente Gammelgaard and Combs, {Gerald F}",
note = "{\textcopyright} 2013.",
year = "2013",
month = dec,
doi = "10.1016/j.jnutbio.2013.04.007",
language = "English",
volume = "24",
pages = "2023--30",
journal = "Journal of Nutritional Biochemistry",
issn = "0955-2863",
publisher = "Elsevier",
number = "12",

}

RIS

TY - JOUR

T1 - Metabolism of selenite to selenosugar and trimethylselenonium in vivo

T2 - tissue dependency and requirement for S-adenosylmethionine-dependent methylation

AU - Jackson, Matthew I

AU - Lunøe, Kristoffer

AU - Gabel-Jensen, Charlotte

AU - Gammelgaard, Bente

AU - Combs, Gerald F

N1 - © 2013.

PY - 2013/12

Y1 - 2013/12

N2 - Impaired S-adenosylmethionine (SAM)-dependent transmethylation and methylation capacity feature in diseases related to obesity or aging, and selenium (Se) metabolism is altered in these states. We tested the hypothesis that SAM metabolism is required for methylation and excretion of Se in a rat model. Four hours after selenite and periodate-oxidized adenosine (POA; an inhibitor of SAM metabolism) were administered, circulating markers of single-carbon status were unchanged, except for decreased circulating phosphatidylcholine (P

AB - Impaired S-adenosylmethionine (SAM)-dependent transmethylation and methylation capacity feature in diseases related to obesity or aging, and selenium (Se) metabolism is altered in these states. We tested the hypothesis that SAM metabolism is required for methylation and excretion of Se in a rat model. Four hours after selenite and periodate-oxidized adenosine (POA; an inhibitor of SAM metabolism) were administered, circulating markers of single-carbon status were unchanged, except for decreased circulating phosphatidylcholine (P

U2 - 10.1016/j.jnutbio.2013.04.007

DO - 10.1016/j.jnutbio.2013.04.007

M3 - Journal article

C2 - 24139672

VL - 24

SP - 2023

EP - 2030

JO - Journal of Nutritional Biochemistry

JF - Journal of Nutritional Biochemistry

SN - 0955-2863

IS - 12

ER -

ID: 104572974